Nanyang Technology University

Academic Profile
Assoc Prof Li Hoi Yeung 

Associate Professor 
Division of Molecular Genetics & Cell Biology 
School of Biological Sciences 
College of Science 



Email: HYLI@ntu.edu.sg
Phone: (+65)63162820/2931 
Office: SBS-01n-15/03n-06 
Education
  • PhD Chinese University of Hong Kong 2000
  • BSc(Hons) Hong Kong Polytechnic University 1997
Biography
Dec 2000 Ph.D. in Biochemistry, The Chinese University of Hong Kong
Aug 2000 – Dec 2004 Postdoctoral Fellow, Howard Hughes Medical Institute, Carnegie Institution of Washington, Baltimore, MD 21210, USA
Dec 2004 – Present Assistant Professor, School of Biological Sciences, Nanyang Technological University, Singapore
Research Interests
1. Regulation of mitotic catastrophe
2. Regulation of spindle assembly and chromosome segregation.
3. Production of the RanGTP gradient in vivo.
4. Functional study of the RanGTPase in cell cycle progression.
5. Regulation of RCC1 during apoptosis
Research Grant
  • A*STAR Biomedical Research Council (2006-)
  • Academic Research Fund Tier 1 (2009-2013)
  • Academic Research Fund Tier 1 (2012-)
  • Academic Research Fund Tier 2 (2006-)
  • College of Science Collaborative Research Award (2011-)
  • Supplementary Equipment Purchase (2006-) [by Ministry of Education (MOE)]
Current Projects
  • A regulatory role of RanGTP in kinetochore-spindle microtubules attachment via Aurora B kinase during metaphase
  • Cell biology analysis and 3D computational modeling of the production of the localized RanGTP concentration and its role in mitotic progression
  • Regulation of RCC1 and its roles in apoptosis
  • Roles of Condensin in regulating mitotic catastrophe
  • Roles of RanGTP in the nuclear accumulatiion of Survivin and its impact on apoptosis
Selected Publications
  • Fung, W.T. Beyzavi, A., Abgrall, P., Nguyen, N.T., and Li, H.Y. (2009). Microfluidic platform for controlling the differentiation of embryoid body,. Lab on a Chip, 9(17), 2591-2595.
  • Ho, C.Y., Wong, C.H. and Li, H.Y. (2008). Perturbation of the chromosomal binding of RCC1, Mad2 and Survivin causes spindle assembly defects and mitotic catastrophe. Journal of cellular biochemistry, 105(3), 835-46.
  • Huang Y.F. Wu M. Li H.Y. (2008). Tumor suppressor ARF promotes non-classic proteasomal independent polyubiquitination of COMMD1. J Biol Chem, In Press(In Press), in Press.
  • Li HY, Ng WP, Wong CH, Iglesias PA, Zheng Y. (2007). Coordination of Chromosome Alignment and Mitotic Progression by the Chromosome-Based Ran Signal. Cell Cycle, .
  • Vong, Q.P.Y., Cao, K., Li, H.Y., Iglesias, P., and Zheng, Y. (2005). Chromosome alignment and segregation regulated by ubiquitination of Survivin. Science, 310, 1499-1504.
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